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Aratac (Amiodarone)
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Amiodarone

A 3-week-old neonate with supraventricular tachycardia unresponsive to traditional therapy was treated successfully with Aratac. At 18-month follow-up, the patient remained euthyroid on Aratac and ventricular arrhythmia free. Intraoperative prophylactic use of Aratac does not prevent new onset of atrial fibrillation in patients undergoing coronary artery bypass grafting and had no effect on outcome.

Other names for this medication:
Amiodarone, Cordarone, Amiodar, Amiodaron, Amiodarona, Ancoron, Aratac, Arycor, Atlansil, Braxan, Cardilor, Cordan, Cordarex, Cornaron, Coronal, Eurythmic, Hexarone, Miodar, Nexterone, Nodis, Opacorden, Pacerone, Pacet, Procor, Sedacoron, Tiaryt, Trangorex,

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Aratac is used to treat and prevent certain types of serious, life-threatening ventricular arrhythmias (a certain type of abnormal heart rhythm when other medications did not help or could not be tolerated. Aratac is in a class of medications called antiarrhythmics. It works by relaxing overactive heart muscles.

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Bone marrow-derived mesenchymal stem cells (BMD-MSCs) are of great interest for tissue engineering, but require expansion before they can be used for therapeutic applications. As a result, such assays are the most popular method for analysing protein-protein interactions, despite being time-consuming and prone to variability.

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A comparison of two popular statistical methods for estimating the time to most recent common ancestor (TMRCA) from a sample of DNA sequences. The aim of this study was to evaluate the effects of metabolic changes on visual parameters during hemodialysis sessions. Thus, our findings provide new insight into the effects of TSLP in metastatic breast cancer.

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CHX10 mutations cause non-syndromic microphthalmia/ anophthalmia in Arab and Jewish kindreds. In particular, estrogen induces structural and functional changes in hippocampal neurons which may contribute significantly to increasing seizure susceptibility. Further studies are necessary to determine the functional implications of the LMP-1 variant on early events in eBL genesis. Myeloperoxidase (MPO) is an enzyme found in myeloid cells, particularly in neutrophils, and to a lesser extent in monocytes and tissue macrophages. Human laboratory research affords an opportunity to investigate validity because one can measure the consistency and sensitivity of pain ratings produced in response to known stimuli.

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Febrile seizures and mechanisms of epileptogenesis: insights from an animal model. Hyperhomocysteinemia activates nuclear factor-kappaB in endothelial cells via oxidative stress. Notch3 and MKP-1 levels are consistently low in dormant tumors, and this is accompanied by relatively high levels of phosphorylated p38, a canonical MKP-1 target previously associated with maintenance of tumor dormancy.

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